Cvičení při CIDP — stupňovaný odpor + aerobik + rovnováha BEZPEČNÉ + zlepšují sílu + 6MWD + únavu (White 2004)

Van den Bergh 2010 + EFNS/PNS 2021 + Van den Bergh 2021 revised + Lewis 2017 + Bunschoten 2019 framework. Chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) is an acquired autoimmune disorder of peripheral nerves — the CHRONIC counterpart to acute Guillain-Barré syndrome (a relapsing or progressive course over >8 weeks distinguishes CIDP from GBS). Classic presentation: SYMMETRIC PROXIMAL AND DISTAL weakness (in contrast to CMT's distal-predominant pattern) + large-fibre sensory loss + reduced or absent reflexes + DEMYELINATION on nerve conduction studies (slow conduction velocities + conduction block + temporal dispersion) + albuminocytologic dissociation in cerebrospinal fluid (high protein with normal cell count). Variants include typical CIDP, Lewis-Sumner / MADSAM (multifocal acquired demyelinating sensory and motor), DADS (distal acquired demyelinating symmetric), sensory-predominant, motor-predominant, and the autoantibody-associated 'nodopathies' (CNTN1, NF155, NF186, CASPR1) — important to recognize because nodopathies may NOT respond to IVIg and need different treatment. CRITICAL DISTINCTION: CIDP is IMMUNOTHERAPY-RESPONSIVE (in stark contrast to CMT, which is hereditary and not immunotherapy-treatable). First-line: IVIg (Hahn 1996 + Hughes 2008 ICE trial), corticosteroids (prednisone or pulse dexamethasone), plasma exchange. Refractory: rituximab + cyclophosphamide + autologous stem cell transplant in selected cases. Diagnostic delay/miss is common — the cost is real (treatable disease, worsens with delayed treatment). Exercise framework per White 2004 + Markvardsen 2018: graded resistance + aerobic + balance training IS safe + IMPROVES strength + 6MWD + balance + reduces fatigue + DOES NOT worsen disease (overturned the cautious 'rest only' framing).

Volya's catalogue carries the foundation moves: supported-glute-bridge for posterior chain low-intensity (proximal weakness pattern), wall-push-up for upper-body push at low load (proximal arm weakness common), scapular-retraction for upper-back posture + shoulder weakness, cat-cow for spinal mobility, supine-knee-to-chest for low-back release + hip flexor stretch, diaphragmatic-breathing for respiratory training (some variants involve respiratory muscles), seated-march for cardio without joint impact + safer than treadmill given sensory loss, ankle-pump for circulation + distal ROM + foot drop counter, calf-raise-rehab for distal-strength preservation + foot drop counter (peripheral demyelination affects distal nerves prominently). The AI coach also knows the nutrition side — vitamin B12 check (deficiency mimics + worsens CIDP, supplement 1000 mcg if low/borderline), copper check if refractory (deficiency mimics CIDP often missed), anti-inflammatory Mediterranean + omega-3, protein 1.2-1.5 g/kg + leucine 2.5-3 g per meal, IVIg-supportive hydration + B12/folate monitoring, steroid-protective low-GI + Ca + vit D + DEXA + B12/folate, infection prevention on immunosuppression (food safety + AVOID unpasteurised + immunocompromised vaccines), AVOID B6 mega-doses >100 mg chronic (paradoxical sensory neuropathy from pyridoxine toxicity), diabetes co-management if present. CRITICAL: GBS/CIDP Foundation International + Foundation for Peripheral Neuropathy + Neuropathy Action Foundation + neurology (CIDP-experienced — KEY because diagnosis often delayed/missed) + nerve conduction studies + B12 + copper + thyroid + glucose deficiency screen + immunology + infusion center if IVIg + dietitian + PT/OT + AFO if foot drop. This is NEVER a replacement for CIDP-experienced multidisciplinary care.